Iodine some useful information about this element that is essential for life. How much iodine does the thyroid gland need? Simple iodine test and dosage regimen

Iodine is a component of T1, T2, T3, T4 hormones, important for pregnant women, before and after childbirth. Iodine is extremely necessary even for healthy people. You will ask iodine for a healthy person, what for? Iodine is safe in high doses. Iodine 131 is poisonous, but potassium iodide is an antagonist of iodine 131 so it counteracts the damaging effects of iodine 131 on the thyroid gland. Our skin can tell how much iodine is needed. Lubricating the thin skin with Lugol. The fluid disappears within minutes, hours and that means iodine needs to be administered. How do I supplement? Orally, you can, but in moderation. Because iodine helats us with bromine which causes undesirable effects. Iodine can be administered by inhalation, e.g. in the form of a bath, or dermally, just like magnesium through the skin, iodine is absorbed very beautifully. A safe dose is 12 µg of potassium iodide, otherwise known as Lugol's solution. Iodine rejuvenates mitochondria, has a beneficial effect in the reproductive systems, prevents infertility, acts in the brain in the prostate, has an anti-inflammatory effect in joint and bone diseases and, most importantly, prevents cancer, especially breast cancer. Before starting iodine dosing, selenium levels should be tested as it is essential for iodine dosing. If the selenium level is normal, you can start administering iodine. I am referring to the words of the respected Polish doctor Andrzej Więckowski.

Iodine deficiency in childhood             ID*- intellectual disability

In ID*, it has been demonstrated that fine motor skills, information processing, and visual problem‐solving are markedly enhanced in school‐attending children following iodine administration. Scientific analysis suggested that ID in mild to moderate levels might prevent children from achieving their full intellectual ability. To some extent, the intelligence quotient frequency distribution in seemingly normal children is shifted toward lower values. This is related to children who did not experience ID in utero because of the correction of deficiency in their mothers before or at the time of initial gestation, which is when ID is most severe.

Iodine deficiency in adulthood

In adults, iodine acts as a significant determinant of thyroid disorders. Hypothyroidism and goiters due to the increase in thyroid activity cause severe ID. In mild to moderate ID, increased thyroid action can compensate for minimal iodine consumption, causing the persistence of euthyroidism in a maximum number of people. Nonetheless, long‐lasting thyroid stimulation causes higher prevalence of these two diseases. This high prevalence of nodular autonomy usually results in a further increase in the prevalence of hyperthyroidism if iodine intake is subsequently increased by salt iodization. Yet, this increase is temporary as iodine adequacy eases thyroid activity.



Bibliography: 
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6145226/